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Toxicologic Pathology
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Commentary: The Utility of Apoptosis Terminology

Richard A. Lockshin

Department of Biological Sciences, St. John's University, Jamaica, New York 11439

References

  • Benson RS, Heer S., Dive C., and Watson AJ (1996). Characterization of cell volume loss in CEM-C7A cells during dexamethasone-induced apoptosis. Am. J. Physiol. 270: C1190-C1203.[Web of Science][Medline] [Order article via Infotrieve]
  • Bortner CD, Hughes FM Jr, and Cidlowski JA (1997). A primary role for K+ and Na+ efflux in the activation of apoptosis. J. Biol. Chem. 272: 32436-32442.[Abstract/Free Full Text]
  • Chen Q., Benson RS, Whetton AD, Brant SR, Donowitz M., Montrose MH, Dive C., and Watson AJ (1997). Role of acid/base homeostasis in the suppression of apoptosis in haemopoietic cells by v-Abl protein tyrosine kinase. J. Cell Sci. 110: 379-387.[Abstract]
  • Gottlieb RA, Gruol DL, Zhu JY, and Engler RL (1996). Preconditioning rabbit cardiomyocytes: Role of pH, vacuolar proton ATPase, and apoptosis. J. Clin. Invest. 97: 2391-2398.[Web of Science][Medline] [Order article via Infotrieve]
  • Gottlieb RA, Nordberg J., Skowronski E., and Babior BM (1996). Apoptosis induced in Jurkat cells by several agents is preceded by intracellular acidification. Proc. Natl. Acad. Sci. USA 93: 654-658.[Abstract/Free Full Text]
  • Ichimiya M., Chang SH, Liu H., Berezesky IK, Trump BF, and Amstad PA (1998). Effect of Bcl-2 on oxidant-induced cell death and intracellular Ca2+ mobilization. Am. J. Physiol. 275: C832-C839.[Web of Science][Medline] [Order article via Infotrieve]
  • Karwatowska-Prokopczuk E., Nordberg JA, Li HL, Engler RL, and Gottlieb RA (1998). Effect of vacuolar proton ATPase on pHi, Ca2+, and apoptosis in neonatal cardiomyocytes during metabolic inhibition/recovery. Circ. Res. 82: 1139-1144.[Abstract/Free Full Text]
  • Levi-Montalcini R. and Aloe L. (1981). Mechanism(s) of action of nerve growth factor in intact and lethally injured sympathetic nerve cells in neonatal rodents. In: Cell Death in Biology and Pathology, ID Bowen and RA Lockshin (eds). Chapman Hall, London, pp. 295-328.
  • Meisenholder GW, Martin SJ, Green DR, Nordberg J., Babior BM, and Gottlieb RA (1996). Events in apoptosis. Acidification is downstream of protease activation and BCL-2 protection. J. Biol. Chem. 271: 16260-16262.[Abstract/Free Full Text]
  • Sikorska M. and Walker PR (1998). Endonuclease activities and apoptosis. In: When Cells Die: A Comprehensive Evaluation of Apoptosis and Programmed Cell Death, RA Lockshin, Z Zakeri, and JL Tilly (eds). Wiley-Liss, New York, pp. 211-242.
  • Trump BF and Berezesky IK (1998). The reactions of cells to lethal injury: Oncosis and necrosis—The role of calcium. In: When Cells Die: A Comprehensive Evaluation of Apoptosis and Programmed Cell Death, RA Lockshin, Z Zakeri, and JL Tilly (eds). Wiley-Liss. New York, pp. 57-96.
  • Zakeri ZF, Quaglino D., Latham T., and Lockshin RA (1993). Delayed intemucleosomal DNA fragmentation in programmed cell death. FASEB J. 7: 470-478.[Abstract]

Toxicologic Pathology, Vol. 27, No. 4, 492-493 (1999)
DOI: 10.1177/019262339902700421


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This Article
Right arrow Free Full Text (Free PDF) Free
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to Saved Citations
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Request Reprints
Right arrow Add to My Marked Citations
Citing Articles
Right arrow Citing Articles via Google Scholar
Right arrow Citing Articles via Scopus
Google Scholar
Right arrow Articles by Lockshin, R. A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Lockshin, R. A.
Social Bookmarking
 Add to CiteULike   Add to Complore   Add to Connotea   Add to Del.icio.us   Add to Digg   Add to Reddit   Add to Technorati   Add to Twitter  
What's this?